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Initially, fracture hem atom a (collection ofblood that surrounds fracture ends of the bones) is formed(Fig 8.26b), and bone necrosis is seen at the ends ofthe frac-tured bone fragments.

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This phosphorylation appears torequire a preceding phosphorylation on serine 15 (Ser15) by several protein kinasesincluding ATM and ATR (reviewed in [41, 88, 86]), which activate p53, but do notstabilize it [93–96].

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